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momo_458新蟲 (初入文壇)
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| 糖尿病時，腎素-血管緊張素系統(tǒng)(renin-angiotensin system，RAS)被激活，升高的血管緊張素Ⅱ(AngⅡ)通過細胞表面的AT1受體，刺激心肌成纖維細胞增生及膠原代謝改變，引起心臟結(jié)構(gòu)重塑，導(dǎo)致心肌間質(zhì)及血管周圍纖維化，膠原含量增多和排列紊亂，造成心室肌僵硬而影響舒張功能，出現(xiàn)糖尿病心肌病(diabetic cardiomyopathy，DCM)的臨床癥狀。本文從RAS的主要成分Ang Ⅱ、Ang-(1-7)、Ac-SDKP和管緊張素受體(ATR)入手，闡述了RAS在糖尿病心肌病發(fā)生發(fā)展中所起的重要作用。 |
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momo_458(金幣+12, 翻譯EPI+1): 2010-08-28 09:57:00
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Diabetes, the renin - angiotensin system (renin-angiotensin system, RAS) is activated, elevated angiotensin Ⅱ (Ang Ⅱ) AT1 receptor by cell surface and stimulate cardiac fibroblast proliferation and collagen metabolism changes cardiac remodeling, resulting in myocardial interstitial and perivascular fibrosis, and disordered collagen content increased, resulting in ventricular stiffness affect diastolic function, heart disease to diabetes (diabetic cardiomyopathy, DCM) of the clinical symptoms. In this paper, the main component of RAS Ang Ⅱ, Ang-(1-7), Ac-SDKP and Angiotensin Receptor (ATR) and expounds the RAS in the development of diabetic cardiomyopathy played an important role. This article comes from: wooden insect Forum http://www.gaoyang168.com/bbs/viewthread.php?tid=2329874&fpage=2 |
2樓2010-08-27 14:16:33
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