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Although a causal connection between the accumulation of chitinase and -1,3-glucanase and systemic protection has not yet been established, the occurrence of high levels of these enzymes in systemically protected tissue suggest their involvement in disease resistance. In fresh apples, the onset of the systemic resistance to B. cinerea closely paralleled the increase in chitinase and -1,3-glucanase activity in systemically protected tissue. These results, which are consistent with the temporal and spatial induction of pathogenesis-related (PR) proteins and likely concomitantly with induced resistance (8,21), show that apple fruit are capable of responding to microbial elicitor treatment as seen in other crops. In several plant–pathogen interactions, the induction and accumulation of PR proteins including chitinase and -1,3-glucanase is often correlated with the onset of induced resistance (12, 17,21,22). Chitinase and -1,3-glucanase hydrolyze fungal cell walls and in combination they have been shown to inhibit the in vitro growth of several pathogenic fungi (18,20). |
禁蟲
| 雖然積累之間的因果關(guān)系幾丁質(zhì)酶和-1,3 - 葡聚糖酶和系統(tǒng)性的保護(hù)尚未建立,這些酶系統(tǒng)保護(hù)組織高水平的發(fā)生,表明其在抗病的參與。新鮮的蘋果,全身抵抗力灰霉病的發(fā)病密切平行的組織系統(tǒng)保護(hù)中的幾丁質(zhì)酶和-1,3 - 葡聚糖酶的活性增加。 ,這是一致的病程相關(guān)(PR)的蛋白質(zhì)的時(shí)空感應(yīng)和隨之而來可能與誘導(dǎo)抗性(8,21),這些結(jié)果表明,蘋果果實(shí)是能夠響應(yīng)其他作物,微生物誘導(dǎo)子處理。在幾種植物鈥損athogen相互作用,誘導(dǎo)和PR蛋白的積累,包括幾丁質(zhì)酶和-1,3 - 葡聚糖酶往往與誘導(dǎo)抗性(12,17,21,22)的發(fā)病密切相關(guān)。幾丁質(zhì)酶和-1,3 - 葡聚糖酶水解真菌細(xì)胞壁和組合,他們已被證明能夠抑制在體外對(duì)多種病原真菌(18,20)增長。 |

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