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yin198716新蟲 (正式寫手)
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| However, tip cells re-express VEGFR3, and its pharmacological inhibition diminishes sprouting. In contrast,loss of VEGFR1 increases sprouting and vascularization. A soluble variant or a kinase-dead mutant of VEGFR1 rescues vascular defects caused by VEGFR1 deficiency, suggesting that this receptor functions as a VEGF trap. VEGFR1 is predominantly expressed in stalk cells and involved in guidance and limiting tip cell formation. |

鐵桿木蟲 (初入文壇)
| 然而,頂端細(xì)胞重新表達(dá)VEGFR3,其藥理上的抑制作用可減弱出芽現(xiàn)象。與此相反,VEGFR1表達(dá)丟失可增加出芽現(xiàn)象和血管形成。VEGFR1可溶性突變體或激酶活性缺失突變體可糾正VEGFR1缺失所造成的缺陷,這表明該受體的功能為收集VEGF。VEGFR1主要表達(dá)于柄細(xì)胞中,并參與引導(dǎo)及限制頂端細(xì)胞的形成。 |
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