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linda831223新蟲 (初入文壇)
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求SCI文章檢索號(hào)
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Compromised autophagy by MIR30B benefits the intracellular survival of Helicobacter pylori. Autophagy. 2012 Jul 1;8(7):1045-57 因無法登陸web of science，求SCI文章檢索號(hào)，填表用，謝謝！ |
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linda831223: 金幣+5, ★★★★★最佳答案, 十分謝謝！ 2014-08-20 12:03:20
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感謝參與，應(yīng)助指數(shù) +1
linda831223: 金幣+5, ★★★★★最佳答案, 十分謝謝！ 2014-08-20 12:03:20
jssxh: 金幣+5, 檢索EPI+1, 謝謝參與，請(qǐng)繼續(xù)關(guān)注本版塊！ 2014-08-21 21:26:05
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Compromised autophagy by MIR30B benefits the intracellular survival of Helicobacter pylori 作者:Tang, B (Tang, Bin)[ 1,3 ] ; Li, N (Li, Na)[ 1 ] ; Gu, J (Gu, Jiang)[ 1 ] ; Zhuang, Y (Zhuang, Yuan)[ 1 ] ; Li, Q (Li, Qian)[ 1 ] ; Wang, HG (Wang, Hai-Guang)[ 1 ] ; Fang, Y (Fang, Yao)[ 1 ] ; Yu, B (Yu, Bo)[ 1 ] ; Zhang, JY (Zhang, Jin-Yu)[ 1 ] ; Xie, QH (Xie, Qing-Hua)[ 1 ] 更多內(nèi)容 AUTOPHAGY 卷: 8 期: 7 頁: 1045-1057 DOI: 10.4161/auto.20159 出版年: JUL 2012 查看期刊信息 摘要 Helicobacter pylori evade immune responses and achieve persistent colonization in the stomach. However, the mechanism by which H. pylori infections persist is not clear. In this study, we showed that MIR30B is upregulated during H. pylori infection of an AGS cell line and human gastric tissues. Upregulation of MIR30B benefited bacterial replication by compromising the process of autophagy during the H. pylori infection. As a potential mechanistic explanation for this observation, we demonstrate that MIR30B directly targets ATG12 and BECN1, which are important proteins involved in autophagy. These results suggest that compromise of autophagy by MIR30B allows intracellular H. pylori to evade autophagic clearance, thereby contributing to the persistence of H. pylori infections. 關(guān)鍵詞 作者關(guān)鍵詞:Helicobacter pylori; MIR30B; autophagy; ATG12; BECN1 KeyWords Plus:GASTRIC EPITHELIAL-CELLS; INFLAMMATORY RESPONSE; MONITORING AUTOPHAGY; MICRORNA EXPRESSION; IMMUNE-RESPONSES; CANCER; INDUCTION; INFECTION; RESISTANCE; APOPTOSIS 作者信息 通訊作者地址: Zou, QM (通訊作者) 顯示增強(qiáng)組織信息的名稱 Third Mil Med Univ, Coll Med Lab Sci, Dept Clin Microbiol & Immunol, Chongqing, Peoples R China. 地址: 顯示增強(qiáng)組織信息的名稱 [ 1 ] Third Mil Med Univ, Coll Med Lab Sci, Dept Clin Microbiol & Immunol, Chongqing, Peoples R China 顯示增強(qiáng)組織信息的名稱 [ 2 ] Chinese Acad Sci, Inst Microbiol, Biol Resource Ctr, Beijing, Peoples R China [ 3 ] Emei Sanat Chengdu Mil Reg, Emeishan, Sichuan, Peoples R China 電子郵件地址:qmzou2007@163.com; mxh95xy@tom.com 基金資助致謝 基金資助機(jī)構(gòu) 授權(quán)號(hào) National Natural Science Foundation of China (NSFC) 81071412 查看基金資助信息 出版商 LANDES BIOSCIENCE, 1806 RIO GRANDE ST, AUSTIN, TX 78702 USA 類別 / 分類 研究方向:Cell Biology Web of Science 類別:Cell Biology 文獻(xiàn)信息 文獻(xiàn)類型:Article 語種:English 入藏號(hào): WOS:000307830600004 ISSN: 1554-8627 期刊信息 Impact Factor (影響因子): Journal Citation Reports® 其他信息 IDS 號(hào): 993BX Web of Science 核心合集中的 "引用的參考文獻(xiàn)": 43 Web of Science 核心合集中的 "被引頻次": 5 |
2樓2014-08-20 11:53:17
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