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| 當基因缺失或敲除時可抑制酸誘導(dǎo)的神經(jīng)元損傷，但轉(zhuǎn)染該基因后又可重建其敏感性.已有研究表明，在未受損的動物腦組織中，僅僅降低PH值至6.5或缺血并不會引起損傷，但當缺血合并酸中毒時，卻可顯著增強毒性作用，導(dǎo)致明顯的腦損傷 |
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Carena
鐵桿木蟲 (知名作家)
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發(fā)芽土豆(金幣+1):Thx for your participation! 2010-07-20 22:38:33
發(fā)芽土豆(金幣+1):Thx for your participation! 2010-07-20 22:38:33
| Gene deletion and gene knock-out can restrain acid-induced neuronal injury. The transfection of the gene would, however, rebuild its sensitivity. Studies have shown that in animals' brain tissues ,which are free of damage, reduction of the PH value to 6.5 or the ischemia will not cause injury. But ischemia and acid, when combined together, would cause intoxication and greatly increase toxic effects , which will result in obvious brain damage. |
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發(fā)芽土豆(金幣+1):Thx for your participation! 2010-07-20 22:39:02
發(fā)芽土豆(金幣+1):Thx for your participation! 2010-07-20 22:39:02
| When the gene knockout or inhibits acid when induced neurons damage, but the genes and transfection rebuilding its sensitivity. Studies have shown that, in the intact animals in the brain, only lower PH value to 6.5 or ischemic damage, but will not cause ischemic merger acidosis, when can significantly increase but the toxic effects, resulting in significantly brain damage |
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| When there is gene deletion or knockout, the acid-induced neuronal damage can be inhibited. The transfection of the gene would, however, rebuild its sensitivity. Studies have shown that in healthy animal brain tissues, neither decrease of the PH value to 6.5 nor ischemia alone causes any danage. But when ischemia and excessive acid are combined together, the toxicity would significantly increase, which results in obvious brain damage. |
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