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蕭肅清揚(yáng)新蟲 (正式寫手)
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monoclonal Abs against tumor-expressed target Ags offer the possibility to re system to combat malignant tumor growth (1). The cruit the innate immune cellular arm of innate immunity comprises immune effector cells,such as monocytes/macrophages, polymorphonuclear cells (PMNs),and NK cells, whereas the complement system constitutes an integral part of the humoral arm (2). Cellular attack mechanisms are triggered by Abs’ Fc regions through interaction with FcRs, expressed on immune effector cells, to mediate Ab-dependent cellmediated cytotoxicity (ADCC) or Ab-dependent cell-mediated phagocytosis.Fixation of the C1q component of the classical complement pathway to target Ag-bound Fc is typically required to induce complement-dependent cytotoxicity (CDC) against target cells |
至尊木蟲 (知名作家)
Translator and Proofreader
| 針對腫瘤表達(dá)的目標(biāo)抗原 (Ags) 而產(chǎn)生的單克隆抗體 (Abs) 為招募先天免疫系統(tǒng)抵御惡性腫瘤的生長提供可能性。先天免疫的組成包括免疫效應(yīng)細(xì)胞,如單核細(xì)胞/巨噬細(xì)胞,多形核細(xì)胞(中性粒細(xì)胞),和NK細(xì)胞;而補(bǔ)體系統(tǒng)構(gòu)成體液免疫的一個組成部分。細(xì)胞攻擊機(jī)制由抗體的可結(jié)晶片段(Fc區(qū))與表達(dá)于免疫效應(yīng)細(xì)胞的FcR相互作用而觸發(fā),介導(dǎo)抗體依賴性細(xì)胞介導(dǎo)的細(xì)胞毒性(ADCC)或抗體依賴細(xì)胞介導(dǎo)的吞噬作用。通常需要將經(jīng)典補(bǔ)體途徑中的補(bǔ)體C1q固著于目標(biāo)抗原結(jié)合的Fc區(qū),以針對靶細(xì)胞誘導(dǎo)補(bǔ)體依賴性細(xì)胞毒性(CDC)。 |
新蟲 (正式寫手)
新蟲 (正式寫手)
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